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**Thirty minutes after this was faxed to Langley AFB, VA, the baldheaded talk show host broke his programming format and for ten minutes denounced anyone who believed in “chemtrails”…

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There was one term that had me puzzled for a minute and that was TOS. Afer a quick search, I was able to find out that it stood for Tactical Operations Support

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SUBJ: "Treaty on Open Skies" (TOS) Aircraft Executing Operational Bio-Chemical Missions Against the United States

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I think TOS (in the letter) is the ackronym he uses for

SUBJ: "Treaty on Open Skies" (TOS) Aircraft Executing Operational Bio-Chemical Missions Against the United States

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An extremely high percentage of our members report the following: Chronic Fatigue Syndrome (CFS), Fibromyalgia (Myalgic Encephalopathy or ME), "brain fog" or cognitive decline, ADHD, mood disorders (primarily Bipolar Disorder and depression), joint swelling and pain, rapid visual and neurological decline, autoimmune disease, and hair loss. Many of these common additional symptoms appear to be related to inflammation.

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The incubation period of LCMV infection is usually between 8 and 13 days. A characteristic biphasic febrile illness then follows. The initial phase, which may last as long as a week, typically begins with any or all of the following symptoms: fever, malaise, anorexia, muscle aches, headache, nausea, and vomiting. Other symptoms that appear less frequently include sore throat, cough, joint pain, chest pain, testicular pain, and parotid (salivary gland) pain. Following a few days of remission, the second phase of the disease occurs, consisting of symptoms of meningitis (for example, fever, headache, and a stiff neck) or characteristics of encephalitis (for example, drowsiness, confusion, sensory disturbances, and/or motor abnormalities, such as paralysis). LCMV has also been known to cause acute hydrocephalus, which often requires surgical shunting to relieve increased intracranial pressure. In rare instances, infection results in myelitis (inflammation of the spinal cord) and presents with symptoms such as muscle weakness, paralysis, or changes in body sensation. An association between LCMV infection and myocarditis (inflammation of the heart muscles) has been suggested.

During the first phase of the disease, the most common laboratory abnormalities are a low white blood cell count (leukopenia) and a low platelet count (thrombocytopenia). Liver enzymes in the serum may also be mildly elevated. After the onset of neurological disease during the second phase, an increase in protein levels, an increase in the number of white blood cells or a decrease in the glucose levels in the cerebrospinal fluid (CSF) is usually found.

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Lymphocytic choriomeningitis, or LCM, is a rodent-borne viral infectious disease that presents as aseptic meningitis (inflammation of the membrane, or meninges, that surrounds the brain and spinal cord), encephalitis (inflammation of the brain), or meningoencephalitis (inflammation of both the brain and meninges). Its causative agent is the lymphocytic choriomeningitis virus (LCMV), a member of the family Arenaviridae, that was initially isolated in 1933. Although LCMV is most commonly recognized as causing neurological disease, as its name implies, asymptomatic infection or mild febrile illnesses are common clinical manifestations. Additionally, pregnancy-related infection has been associated with abortion, congenital hydrocephalus and chorioretinitis, and mental retardation.

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Background: Lymphocytic choriomeningitis virus (LCMV) is a single-stranded RNA virus of the family Arenaviridae. Other members of Arenaviridae include Lassa, Argentine hemorrhagic fever, Bolivian hemorrhagic fever, and Venezuelan hemorrhagic fever viruses. LCMV causes an asymptomatic infection in its rodent reservoir (eg, mice) and has variable effect on hamster health. It is passed to humans when they inhale, consume, or otherwise contact infected excreta. Typically a febrile, self-limited, biphasic disease, LCMV is a significant teratogen whose role in congenital infections has only recently gained appreciation. Individuals who are exposed to rodents from living conditions or other exposure, such as pet handlers and laboratory personnel, are at particular risk for the infection.

Pathophysiology: After transmission of LCMV via inhalation, consumption, or cutaneous or mucosal exposure, a primary viremia ensues, resulting in extra–central nervous system (CNS) seeding and an initial, nonspecific, febrile illness. Several days later, secondary viremia follows and frequently results in CNS involvement and typical meningitis/meningoencephalitis. The manifestations of the disease are thought to result from the host response to the virus as natural killer cells; then, cytotoxic T cells respond to infection with the production of interferon and additional inflammatory mediators. No human-to-human transmission occurs, except in congenital disease. Congenital LCMV infection is typically much more serious than the acquired disease. It mimics toxoplasmosis and cytomegalovirus (CMV) congenital infection. As in acquired infection, congenital disease is thought to result from T- and B-cell–mediated injury to the fetal host.

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Mortality/Morbidity: Fewer than 1% of patients with acquired infection have fatal disease, usually encephalitis or massive hemorrhage. By contrast, 35% of congenital infections are believed to result in fetal loss. Most surviving infants (84%) have neurodevelopmental sequelae, including cerebral palsy, seizures, decreased visual acuity, mental retardation, and progressive hydrocephalus.

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How is LCMV spread, and how do humans become infected?

LCMV is naturally spread by the common house mouse, Mus musculus. Once infected, these mice can become chronically infected by maintaining virus in their blood and/or persistently shedding virus in their urine, a common characteristic of other arenavirus infections in rodents. Chronically infected female mice usually transmit infection to their offspring, which in turn become chronically infected.

Humans become infected by inhaling infectious aerosolized particles of rodent urine, feces, or saliva, by ingesting food contaminated with virus, by contamination of mucus membranes with infected body fluids, or by directly exposing cuts or other open wounds to virus-infected blood. LCMV infection has also been documented among staff handling infected hamsters. Person-to-person transmission has not been reported, with the exception of vertical transmission from an infected mother to fetus.

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