posted 02-28-2002 04:05 AM            

Source: http://www.hummelcroton.com/m_bastea.html

===========================

BARIUM STEARATE

HUMMEL CROTON INC.
10 HARMICH ROAD
BUSINESS HRS.: 908-754-1800
SOUTH PLAINFIELD, NJ O7080

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I. PRODUCT IDENTIFICATION:
--------------------------------------------------------------------------------

PRODUCT NAME........: Barium Stearate

CHEMICAL FAMILY.....:
Barium Salt of Stearic Aci0d

CHEMICAL NAME.......: Barium Stearate

CAS NUMBER..........: CAS # 6865-35-6

FORMULA.............: Ba(C18H35O2)2

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II. HAZARDOUS INGREDIENTS:
--------------------------------------------------------------------------------

INGREDIENT NAME
Barium Stearate

CAS # : 6865-35-6

EXPOSURE LIMITS
OSHA : Not Established
ACGIH: Not Established

CONCENTRATION (%)
Essentially 100 %

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III. PHYSICAL PROPERTIES:
--------------------------------------------------------------------------------

PHYSICAL FORM.............: Powder

COLOR.....................: Cream to white

ODOR......................: Odorless

MOLECULAR WEIGHT..........: 704,28

pH .......................: Neutral

BOILING POINT.............: Not Applicable

MELTING/FREEZING POINT....: 120oC

VISCOSITY.................: Not Applicable

SOLUBILITY IN WATER ......: Negligible

SPECIFIC GRAVITY .........: 1.23

BULK DENSITY..............: Not Established

VAPOR PRESSURE ...........: Not Volatile

VAPOR DENSITY ............:
Not Applicable (Air = 1)

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IV. FIRE AND EXPLOSION DATA:
--------------------------------------------------------------------------------

FLASH POINT.....................:
Not Applicable.

AUTO-IGNITION TEMPERATURE.......:
Not Established

EXTINGUISHING MEDIA.............:
Water Spray, Foam, Carbon Dioxide
0or Dry Chemical

SPECIAL FIRE FIGHTING PROCEDURES:
Burning will produce toxic fumes.
Wear self-contained breathing apparatus
and full turnout gear to fight fires.

UNUSUAL FIRE / EXPLOSION HAZARDS:
Concentrated dust may present an
explosion hazard.

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V. HUMAN HEALTH DATA:
--------------------------------------------------------------------------------

ROUTE(S) OF ENTRY.......:
Eye Contact; Skin Contact; Inhalation;
Ingestion

HUMAN EFFECTS AND SYMPTOMS OF OVEREXPOSURE:

ACUTE INHALATION..............:
Inhalation may irritate the respiratory tract.
Symptoms may include coughing, shortness of
breath, sore throat and runny nose. If
sufficient amounts are inhaled and
absorbed, symptoms may resemble those in acute
ingestion.

ACUTE SKIN CONTACT............:
Skin contact may cause irritation with
symptoms of redness, swelling, itching
and pain.

ACUTE EYE CONTACT.............:
Eye contact may cause irritation with symptoms
of redness, swelling, itching, tearing
and pain.

ACUTE INGESTION...............:
Ingestion may cause gastroenteritis
(inflammation of the lining membrane of
the stomach and intestines) with abdominal
pain, nausea, vomiting and diarrhea. Systemic
effects may follow and may include ringing of
the ears, dizziness, elevated blood pressure,
blurred vision and tremors.

CHRONIC EFFECTS OF EXPOSURE...:

CARCINOGENICITY...............:
This product is not listed by NTP, IARC or
regulated as a carcinogen by OSHA.

MEDICAL CONDITIONS AGGRAVATED BY EXPOSURE..:
Persons with preexisting eye or skin
conditions or impaired pulmonary function may
be more susceptible to the effects of
this product.

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VI. EMERGENCY AND FIRST AID PROCEDURES:
--------------------------------------------------------------------------------

FIRST AID FOR EYES......:
In case of contact, immediately flush eyes
with plenty of water for at least 15 minutes.
Call a physician.

FIRST AID FOR SKIN......:
In case of contact, flush skin with water.
Wash clothing before reuse. Call a physician
if irritation occurs.

FIRST AID FOR INHALATION:
If inhaled, remove to fresh air. If not
breathing, give artificial respiration. If
breathing is difficult, give oxygen.
Call a physician.

FIRST AID FOR INGESTION.:
If swallowed, call a physician
immediately.

--------------------------------------------------------------------------------
VII. EMPLOYEE PROTECTION RECOMMENDATIONS:
--------------------------------------------------------------------------------

EYE PROTECTION REQUIREMENTS........:
Safety glasses or goggles.

SKIN PROTECTION REQUIREMENTS.......:
PVC gloves with impervious boots, apron or
coveralls. Employees should wash their hands
and face before eating, drinking or using
tobacco products.

RESPIRATOR REQUIREMENTS............:
Work ambient concentrations should be
monitored and if the recommended exposure
limit is exceeded, a NIOSH/MSHA approved dust
respirator must be worn.

VENTILATION REQUIREMENTS...........:
Use local ventilation if dusting is a problem,
to maintain air levels below the recommended
exposure limit.

ADDITIONAL PROTECTIVE MEASURES.....:
Emergency showers and eye wash stations
should be available. Educate and train
employees in the safe use and handling
of hazardous chemicals.

--------------------------------------------------------------------------------
VIII. REACTIVITY DATA:
--------------------------------------------------------------------------------

STABILITY..................:
Stable under ordinary conditions of
use and storage.

HAZARDOUS POLYMERIZATION...: Will not occur.

INCOMPATIBILITIES..........: Strong Oxidizers

INSTABILITY CONDITIONS.....:
Excessive temperatures
(see INCOMPATIBILITIES).

DECOMPOSITION PRODUCTS.....:
Barium Oxide, Carbon Dioxide,
Carbon Monoxide and Water.

--------------------------------------------------------------------------------
IX. SPILL AND LEAK PROCEDURES:
--------------------------------------------------------------------------------

SPILL OR LEAK PROCEDURES....:
Utilize recommended protective clothing and
equipment. Clean spills in a manner that does
not disperse dust into the air. Spill area can
be washed with water. Collect wash water for
approved disposal. Keep from entering water
or ground water.

WASTE DISPOSAL METHOD.......:
Waste disposal should be in accordance with
existing federal, state and local
environmental regulations.

--------------------------------------------------------------------------------
X. SPECIAL PRECAUTIONS & STORAGE DATA:
--------------------------------------------------------------------------------

STORAGE TEMPERATURE(MIN/MAX): Ambient/Ambient.

SHELF LIFE..................:
Unlimited in tightly closed container.

SPECIAL SENSITIVITY.........: None

HANDLING/STORAGE PRECAUTIONS:
Avoid breathing dust. Avoid getting in eyes
or on skin. Wash thoroughly after handling.
Store in a dry place away from direct
sunlight, heat and incompatible materials
(see Section VIII). Reseal containers
immediately after use. Store away from food
and beverages.

--------------------------------------------------------------------------------
XI. SHIPPING INFORMATION:
--------------------------------------------------------------------------------

Contact Hummel Croton for
current shipping Information

--------------------------------------------------------------------------------
XII. TOXICITY DATA:
--------------------------------------------------------------------------------

To the best of our knowledge, the chemical,
physical, and toxicological properties have
not been thoroughly investigated for
Barium Stearate.

The information for Stearic acid is as follows:

STEARIC ACID

IRRITATION DATA

SKN-HMN 75 MG/3D-I MLD
85DKA8 -,127,77

SKN-RBT 500 MG/24H MOD
FCTXAV 17,383,79

TOXICITY DATA

IVN-RAT LD50:21500 UG/KG
APTOA6 18,141,61

IVN-MUS LD50:23 MG/KG
APTOA6 18,141,61

SKN-RBT LD50:>5 GM/KG
FCTXAV 17,383,79

TARGET ORGAN DATA

Behavioral (convulsions or effect on seizure
threshold) lungs, thorax or respiration
(other changes)

kidney, ureter, bladder (bladder tumors)
tumorigenic (equivocal tumorigenic agent
by RTECS criteria)

only selected registry of toxic effects
of chemical substances(RTECS) data is
presented here. See actual entry in RTECS
for complete information.

--------------------------------------------------------------------------------
XIII. REGULATORY INFORMATION:
--------------------------------------------------------------------------------

OSHA STATUS.................:
This product is hazardous under the criteria
of the Federal OSHA Hazard Communication
Standard 29 CFR 1910.1200.

TSCA STATUS.................:
On TSCA Inventory

RCRA STATUS.................:
If discarded in its purchased form, this
product would not be hazardous waste either
by listing or by characteristic. However,
under RCRA, it is the responsibility of the
product user to determine at the time of
disposal, whether a material containing the
product or derived from the product should be
classified as a hazardous waste.
(40 CFR 261.20-24)

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XIV. ECOLOGICAL INFORMATION:
--------------------------------------------------------------------------------

Data not Available at the present time.

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XV. APPROVALS:
--------------------------------------------------------------------------------

REASON FOR ISSUE..........:
Update to new format.

PREPARED BY...............: Mark Dugan

APPROVED BY...............: Mark Dugan

posted 02-28-2002 07:11 AM            

A number of accidental barium poisonings have occurred following the ingestion of barium salts. The acute symptoms include excess salivation, vomiting, diarrhea, increased blood pressure, muscular tremors, weakness, paresis, anxiety, dyspnea, and cardiac irregularities. A severe loss of potassium can account for some of the symptoms. Convulsions and death from cardiac and respiratory failure can occur. Magnesium and sodium sulfate are antidotal if taken soon after ingestion since either salt will result in the formation of insoluble barium sulfate and prevent further absorption. http://risk.lsd.ornl.gov/tox/profiles/barium_f_V1.shtml

posted 02-28-2002 12:11 PM            

The clouds illuminated by the moon last night were reddish. I feel like I'm living in a chemical waste dump. I wish those zombies in the military had more compassion for the people and environment they are poisoning.

posted 02-28-2002 07:02 PM            

I can sympathize with your irritated eyes. I’ve had reddened eyes off and on for some time which I could not explain. I think I understand why now due to these particles. Having to flush one’s eyes after having chemtrails evident is inconvenient, but better to know what to do than not. I’ve also suffered from chronic sinus problems since 1999 and I am now wondering if they may be related to chemtrails.

If you are especially allergic to things, I suggest you try a liver/gallstone cleanse to get out gall/liver stones naturally. Some people have said these cleanses reduce their allergies in general due to better functionality of the liver. Hulda Clark is the biggest proponent of the liver cleanse. I’ve done it twice and gotten out many ‘stones’. The cleanse is at http://www.drclark.net/info/liver.htm. Her book is “The Cure for all Diseases”.

posted 02-28-2002 11:04 PM            

ALUMIMUM POWDERS

HUMMEL CROTON INC.
10 HARMICH ROAD
BUSINESS HRS.: 908-754-1800
SOUTH PLAINFIELD, NJ O7080

EMERGENCY PHONE NUMBERS
BUSINESS HRS.: 908-754-1800
CHEMTREC: 800-424-9300

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I. PRODUCT IDENTIFICATION:
--------------------------------------------------------------------------------

PRODUCT NAME........: Aluminum

CHEMICAL FAMILY.....: Metal Powder

CHEMICAL NAME.......: Aluminum

SYNONYMS............: Aluminum

CAS NUMBER..........: CAS #: 7429-90-5

FORMULA.............: Al

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II. HAZARDOUS INGREDIENTS:
--------------------------------------------------------------------------------
INGREDIENT NAME
Aluminum

CAS # 7429-90-5

EXPOSURE LIMITS
OSHA : 15 MG(AL)/M3, Total Dust
ACGIH: 10 MG/M3, Dust

CONCENTRATION (%)
Essentially 100 %

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III. PHYSICAL PROPERTIES:
--------------------------------------------------------------------------------

PHYSICAL FORM.............: Solid, fine
powder or granular material

COLOR.....................: shinny gray

ODOR......................: Odorless

MOLECULAR WEIGHT..........: 26.98

pH .......................: Not Applicable

BOILING POINT.............: 2327 oC

MELTING/FREEZING POINT....: 660 oC

VISCOSITY.................: Not Applicable

SOLUBILITY IN WATER ......: Not Soluble

SPECIFIC GRAVITY .........: 2.70

BULK DENSITY..............: Not Established

VAPOR DENSITY ............: Not Applicable
(Air = 1)

--------------------------------------------------------------------------------
IV. FIRE AND EXPLOSION DATA:
--------------------------------------------------------------------------------

FLASH POINT.....................:
Not Applicable.

AUTO-IGNITION TEMPERATURE.......: 1400 oF
(759 oC)

EXTINGUISHING MEDIA: Dry Chemical Powder.

SPECIAL FIREFIGHTING PROCEDURES: Wear
Self-Contained Breathing Apparatus And
Protective

Clothing To Prevent Contact With Skin
And Eyes

UNUSUAL FIRE AND EXPLOSIONS HAZARDS:
Emits Toxic Fumes Under
Fire Conditions. Do Not Expose To Air
And Fire. This Material, Like Most
Materials In Powder Form, Is Capable
Of Creating A Dust Explosion.

--------------------------------------------------------------------------------
V. HUMAN HEALTH DATA:
--------------------------------------------------------------------------------

ROUTE(S) OF ENTRY.......: Eye Contact; Skin
Contact; Inhalation; Ingestion

HUMAN EFFECTS AND SYMPTOMS OF OVEREXPOSURE:

ACUTE INHALATION..............: Inhalation
may irritate the respiratory tract.
Symptoms may include coughing, shortness
of breath, sore throat and runny nose. If
sufficient amounts are inhaled and absorbed,
symptoms may resemble those in acute
ingestion.

ACUTE SKIN CONTACT............: Skin contact
may cause irritation with symptoms of
redness, swelling, itching and pain.

ACUTE EYE CONTACT.............: Eye contact
may cause irritation with symptoms of
redness, swelling, itching, tearing and pain.

ACUTE INGESTION...............: Ingestion
may cause gastroenteritis (inflammation of
the lining membrane of the stomach and
intestines) with abdominal pain, nausea,
vomiting and diarrhea. Systemic effects
may follow and may include ringing of the
ears, dizziness, elevated blood pressure,
blurred vision and tremors.

CHRONIC EFFECTS OF EXPOSURE...:

CARCINOGENICITY...............: This product
is not listed by NTP, IARC or regulated as a
carcinogen by OSHA.

MEDICAL CONDITIONS AGGRAVATED BY EXPOSURE...:
Persons with preexisting eye or skin
conditions or impaired pulmonary function may
be more susceptible to the effects of this
product.

--------------------------------------------------------------------------------
VI. EMERGENCY AND FIRST AID PROCEDURES:
--------------------------------------------------------------------------------

FIRST AID FOR EYES......: In case of contact,
immediately flush eyes with plenty of water
for at least 15 minutes. Call a physician.

FIRST AID FOR SKIN......: In case of contact,
flush skin with water. Wash clothing before
reuse. Call a physician if irritation
occurs.

FIRST AID FOR INHALATION: If inhaled, remove
to fresh air. If not breathing, give
artificial respiration. If breathing is
difficult, give oxygen. Call a physician.

FIRST AID FOR INGESTION.: If swallowed, call
a physician immediately.

--------------------------------------------------------------------------------
VII. EMPLOYEE PROTECTION RECOMMENDATIONS:
--------------------------------------------------------------------------------

EYE PROTECTION REQUIREMENTS........:
Safety glasses or goggles.

SKIN PROTECTION REQUIREMENTS.......:
PVC gloves with impervious boots, apron or
coveralls. Employees should wash their hands
and face before eating, drinking or using
tobacco products.

RESPIRATOR REQUIREMENTS............:
Work ambient concentrations should be
monitored and if the recommended exposure
limit is exceeded, a NIOSH/MSHA approved
dust respirator must be worn.

VENTILATION REQUIREMENTS...........:
Use local ventilation if dusting is a
problem, to maintain air levels below the
recommended exposure limit.

ADDITIONAL PROTECTIVE MEASURES.....:
Emergency showers and eye wash stations
should be available. Educate and train
employees in the safe use and handling
of hazardous chemicals.

--------------------------------------------------------------------------------
VIII. REACTIVITY DATA:
--------------------------------------------------------------------------------

STABILITY..................: Stable under
ordinary conditions of use and storage.

HAZARDOUS POLYMERIZATION...: Will not occur.

INCOMPATIBILITIES..........: acids, acid
chlorides, oxidizing agents, halogens, air
sensitive, moisture sensitive

INSTABILITY CONDITIONS.....:
Excessive temperatures
(see INCOMPATIBILITIES).

DECOMPOSITION TEMPERATURE..: Not Established

DECOMPOSITION PRODUCTS.....: toxic fumes of:
aluminum oxide

--------------------------------------------------------------------------------
IX. SPILL AND LEAK PROCEDURES:
--------------------------------------------------------------------------------

SPILL OR LEAK PROCEDURES....: Utilize
recommended protective clothing and equipment.
Clean spills in a manner that does not
disperse dust into the air. Spill area can be
washed with water. Collect wash water for
approved disposal. Keep from entering water
or ground water.

WASTE DISPOSAL METHOD.......: Waste disposal
should be in accordance with existing federal,
state and local environmental regulations.

--------------------------------------------------------------------------------
X. SPECIAL PRECAUTIONS & STORAGE DATA:
--------------------------------------------------------------------------------

STORAGE TEMPERATURE(MIN/MAX): Ambient/Ambient.

SHELF LIFE..................: Unlimited in
tightly closed container.

SPECIAL SENSITIVITY.........: None

HANDLING/STORAGE PRECAUTIONS: Avoid breathing
dust. Avoid getting in eyes or on skin.
Wash thoroughly after handling. Store in a
dry place away from direct sunlight, heat and
incompatible materials (see Section VIII).
Reseal containers immediately after use.
Store away from food and beverages.

--------------------------------------------------------------------------------
XI. SHIPPING INFORMATION:
--------------------------------------------------------------------------------

Contact Hummel Croton for current
shipping Information

--------------------------------------------------------------------------------
XII. TOXICITY DATA:
--------------------------------------------------------------------------------

ACUTE EFFECTS
Harmful if inhaled. May be harmful
if swallowed. May cause irritation.
To the best of our knowledge, the
chemical, physical, and toxicological
properties have not been thoroughly
investigated.

RTECS #: BD0330000

ALUMINUM
only selected registry of toxic effects
of chemical substances

(RTECS) data is presented here. See actual
entry in RTECS for complete information.

--------------------------------------------------------------------------------
XIII. REGULATORY INFORMATION:
--------------------------------------------------------------------------------

OSHA STATUS.................: This product is
hazardous under the criteria of the Federal
OSHA Hazard Communication Standard 29
CFR 1910.1200.

posted 03-01-2002 02:29 AM            

Yes, I felt better after each cleanse. I probably got out around 25 pea sized stones per cleanse along with a lot of smaller 'chaff'. I got out a stone about 3/4 inches in diameter in the second cleanse. I could not believe it. I am reasonably athletic and generally in good shape. I am currently doing a kidney cleanse.

The liver produces up to a quart of bile which exits to the colon via the gall bladder. If the gall bladder is filled with stones the plumbing can get backed up with all sorts of associated health problems! Also, the stones can be backed up in the liver in the bile ducts besides the gall bladder.

posted 03-19-2002 12:05 AM            

The soluble salts of barium, an alkaline earth metal, are toxic in mammalian systems. They are absorbed rapidly from the gastrointestinal tract and are deposited in the muscles, lungs, and bone. Barium is excreted primarily in the feces.

At low doses, barium acts as a muscle stimulant and at higher doses affects the nervous system eventually leading to paralysis. Acute and subchronic oral doses of barium cause vomiting and diarrhea, followed by decreased heart rate and elevated blood pressure. Higher doses result in cardiac irregularities, weakness, tremors, anxiety, and dyspnea. A drop in serum potassium may account for some of the symptoms. Death can occur from cardiac and respiratory failure. Acute doses around 0.8 grams can be fatal to humans.

Subchronic and chronic oral or inhalation exposure primarily affects the cardiovascular system resulting in elevated blood pressure. A lowest-observed-adverse-effect level (LOAEL) of 0.51 mg barium/kg/day based on increased blood pressure was observed in chronic oral rat studies (Perry et al. 1983), whereas human studies identified a no-observed-adverse-effect level (NOAEL) of 0.21 mg barium/kg/day (Wones et al. 1990, Brenniman and Levy 1984). The human data were used by the EPA to calculate a chronic and subchronic oral reference dose (RfD) of 0.07 mg/kg/day (EPA 1995a,b). In the Wones et al. study, human volunteers were given barium up to 10 mg/L in drinking water for 10 weeks. No clinically significant effects were observed. An epidemiological study was conducted by Brenniman and Levy in which human populations ingesting 2 to 10 mg/L of barium in drinking water were compared to a population ingesting 0 to 0.2 mg/L. No significant individual differences were seen; however, a significantly higher mortality rate from all combined cardiovascular diseases was observed with the higher barium level in the 65+ age group. The average barium concentration was 7.3 mg/L, which corresponds to a dose of 0.20 mg/kg/day. Confidence in the oral RfD is rated medium by the EPA.

Subchronic and chronic inhalation exposure of human populations to barium-containing dust can result in a benign pneumoconiosis called "baritosis." This condition is often accompanied by an elevated blood pressure but does not result in a change in pulmonary function. Exposure to an air concentration of 5.2 mg barium carbonate/m3 for 4 hours/day for 6 months has been reported to result in elevated blood pressure and decreased body weight gain in rats (Tarasenko et al. 1977). Reproduction and developmental effects were also observed. Increased fetal mortality was seen after untreated females were mated with males exposed to 5.2 mg/m3 of barium carbonate. Similar results were obtained with female rats treated with 13.4 mg barium carbonate/m3. The NOAEL for developmental effects was 1.15 mg/m3 (equivalent to 0.8 mg barium/m3). An inhalation reference concentration (RfC) of 0.005 mg/m3 for subchronic and 0.0005 mg/m3 for chronic exposure was calculated by the EPA based on the NOAEL for developmental effects (EPA 1995a). These effects have not been substantiated in humans or other animal systems.

Barium has not been evaluated by the EPA for evidence of human carcinogenic potential (EPA 1995b).

3.1 ORAL EXPOSURES
3.1.1 Acute Toxicity
3.1.1.1 Human
A number of accidental barium poisonings have occurred following the ingestion of barium salts. The estimated fatal dose of barium carbonate, a rodenticide, is about 5 grams for a 70 kg human (Arena 1979). The LD50 for barium chloride is estimated at about 1 gram for a 70 kg human (Machata 1988), and the LDLo (lowest published lethal dose) is reported to be about 0.8 grams (Lewis and Sweet 1984). The acute symptoms include excess salivation, vomiting, diarrhea, increased blood pressure, muscular tremors, weakness, paresis, anxiety, dyspnea, and cardiac irregularities. A severe loss of potassium can account for some of the symptoms. Convulsions and death from cardiac and respiratory failure can occur. Magnesium and sodium sulfate are antidotal if taken soon after ingestion since either salt will result in the formation of insoluble barium sulfate and prevent further absorption. Survival for more than 24 hours is usually followed by complete recovery (Arena 1979).

Complications occurred in a woman following a barium swallow investigation for severe dysphagia. Direct aspiration of a large amount of barium into the right main bronchus resulted in tachycardia, tachypnoea, fever, and an oxygen saturation of 82%; two weeks later the woman still had a moist cough with widespread rales but continued to recover (Penington 1993).

A family was accidentally poisoned with barium from eating their evening meal. The mother had fried fish breaded with a flour-like substance that turned out to be rat poison containing barium carbonate. All seven family members, aged 2 to 48 years, developed nausea, vomiting, diarrhea, and crampy abdominal pain within minutes of consuming the meal; the parents also developed ventricular tachycardia, flaccid paralysis of the extremities, shortness of breath (mother), and respiratory failure (father). Patients were treated symptomatically and all fully recovered (Johnson and VanTassell 1991).

3.2 INHALATION EXPOSURES
3.2.1 Acute Toxicity

3.2.1.1 Human
Barium carbonate dust has been reported to be a bronchial irritant. Barium oxide dust is considered a dermal and nasal irritant (Beliles 1994). The effect of barium dusts on welders was investigated under simulated working conditions over a one-week time period (Zschiesche et al. 1992). Barium fume concentrations were 4.4 and 2.0 mg/m3 during welding with stick electrodes and flux cored wires, respectively. No adverse health effects on the welders were attributable to barium exposure, but there was a slight decrease in plasma potassium levels at the end of the work shift.

posted 03-19-2002 12:14 AM            

From 1987 to 1993, according to the Toxics Release Inventory barium compound releases to land and water totaled over 57 million lbs. These releases were primarily from copper smelting industries. The largest releases occurred in Arizona and Utah. The largest direct releases to water occurred in Texas.

What happens to Barium when it is released to the environment? In water, the more toxic soluble barium salts are likely to be converted to insoluble salts which precipitate. Barium does not bind to most soils and may migrate to ground water. It has a low tendency to accumulate in aquatic life.

Effect:
Short-term: EPA has found barium to potentially cause the following health effects when people are exposed to it at levels above the MCL for relatively short periods of time: gastrointestinal disturbances and muscular weakness.

Long-term: Barium has the potential to cause the following effects from a lifetime exposure at levels above the MCL: high blood pressure.

Followup:

Treatment:
Ion Exchange, Reverse Osmosis, Lime Softening, Electrodialysis.
http://www.h2otest.com/factsheets/barium.html

posted 04-19-2002 02:39 AM            

I am very glad that you are taking the initiative to do the cleanses. Wait until you do the liver flush, you will not be disappointed. I will do my 3rd soon. My favorite supplier of black walnut extract, wormwood, and cloves is http://www.shrc.net, run by Hulda Clark's son.

I also recommend you look into the health protocol advanced by Bob Beck. You can do an Internet search on his name and find lots of information. Specifically beneficial is blood electrification and drinking homemade colloidal silver, but all four elements of the protocol are beneficial. The primary supplier of these instruments is Sota Instruments in Canada. It's the only supplier endorsed by Dr. Beck. www.sotainstruments.com. I have bought and used some of their instruments and find them very reliable. Such a company cannot exist in the USA at this time due to undue scruitiny by the FDA and FTC. See also www.sharinghealth.com for information.

posted 04-19-2002 07:54 PM            

This seems to be one that NO debunker, skeptic, etc. seems to want to touch.

posted 04-19-2002 09:27 PM            

Aluminum Toxicity in Infants and Children (RE9607)

AMERICAN ACADEMY OF PEDIATRICS

Committee on Nutrition

Although aluminum is the most abundant metal in the earth's crust and is ubiquitous in its distribution, it has no known useful biological function. Even though the element is present in small amounts in mammalian tissues, its toxic effect on living organisms has become clear only recently. Aluminum is now being implicated as interfering with a variety of cellular and metabolic processes in the nervous system and in other tissues.

ALUMINUM EXPOSURE

Humans are exposed to aluminum from a variety of environmental sources. Because aluminum sulfate (alum) is used as a flocculating agent in the purification of municipal water supplies, drinking water may contain high levels of aluminum. Aluminum cans, containers, and cooking utensils, as well as medications that contain aluminum, are also potential sources of exposure.[1]

Although an increase in body stores of aluminum as a result of transfer through the skin is probably negligible, exposure is common from the use of deodorants containing aluminum. Aluminum inhaled from dust is retained in pulmonary tissue and peribronchial lymph nodes but is largely excluded from other tissues. The aluminum concentration in pulmonary tissues does not correlate with that in other tissues.

The average dietary intake of aluminum by adults is probably 3 to 5 mg/d, of which about 15 mug is absorbed.[2] Most of the aluminum absorbed from the intestinal tract is excreted in urine, leaving total body aluminum stores of less than 30 to 40 mg. Individuals with normal glomerular filtration rates who increase their aluminum intake by ingesting aluminum-containing antacids increase their absorption and urinary excretion of the metal.[3] Healthy individuals seem capable of increasing their renal aluminum clearance from approximately 5% to about 50% of the glomerular filtration rate.[4,5] The low aluminum clearance normally present is largely related to the plasma binding of aluminum by a saturable plasma component at blood levels less than 200 mug/L.[5]

Aluminum levels in tissue are generally low in adults with normal renal function who have ingested large amounts of aluminum-containing antacids for years [4]; however, elevated plasma aluminum levels have been reported in healthy infants given aluminum-containing antacids.[6] In contrast, in patients with chronic renal failure, total body aluminum can be markedly increased from the ingestion of antacids containing aluminum. Bone and liver are the tissues most frequently affected by increased absorption and/or decreased clearance of aluminum.

POTENTIAL FOR TOXICITY
Renal Disease

In 1976, it was reported that the brain tissue of patients dying of a neurologic syndrome called dialysis encephalopathy had high concentrations of aluminum in the gray matter.[7] Two years later, a severe form of osteomalacic osteodystrophy (fracturing dialysis osteodystrophy) and dialysis encephalopathy was described, which occurred in patients undergoing dialysis with a dialysate prepared from tap water that contained large amounts of aluminum.[8] The epidemic-like occurrence of these diseases was largely eliminated by removing the aluminum from the water used to prepare the dialysate.

The first reports of pediatric patients with progressive encephalopathy similar to dialysis encephalopathy described some children who had not received dialysis at the time their symptoms first appeared.[9,10] The children had congenital renal disease and had received doses of aluminum-containing phosphate binders as high as 240 to 800 mg/kg per day for 4 to 12 months. Aluminum toxicity also has been reported from bladder irrigation with aluminum sulfate.[11] Many of these studies do not include data on aluminum levels in plasma and tissue.

A report in 1984 described three infants with azotemia in whom aluminum intoxication developed after treatment with aluminum hydroxide.[12] Biopsies of the iliac crest demonstrated severe osteomalacia and massive deposition of aluminum in the bone. In the same year, another child who had chronic renal failure and normal neurologic findings at 2 years of age was described.[13] This child developed encephalopathy by 8 years of age. The child had not received dialysis but had received aluminum-containing phosphate binders for 6 years. High concentrations of aluminum were found in serum samples and bone biopsy specimens. Other studies have confirmed that children with chronic renal failure who receive aluminum-containing antacids for control of hyperphosphatemia have increased serum aluminum concentrations and bone aluminum levels.[14-16] The data from these studies show a direct correlation between the oral aluminum dose and plasma aluminum concentrations. Plasma aluminum levels greater than 100 mug/L put individuals at risk for aluminum toxicity.[2,12,15] The precise threshold for toxicity is not known, but it may be lower than 100 mug/L.

Alternatives to treatment with aluminum-containing phosphate binders are available. Calcium carbonate has been shown to be superior to aluminum binders in the control of secondary hyperparathyroidism in adults and children with chronic renal failure.[17] Calcium acetate also has been shown to be a safe, effective binder of phosphate.[18] Calcium citrate should not be used as a phosphate binder, because citrate enhances aluminum absorption from dietary sources.[19]

One study has shown that aluminum does not accumulate in infants with chronic renal failure who are not exposed to aluminum-containing antacids or contaminated intravenous solutions.[20]

Intravenous Therapy

High concentrations of aluminum have been found in the bone, urine, and plasma of infants receiving intravenous therapy.[21,22] It has been shown that commercial albumin solutions and a number of substances frequently used as additives in the parenteral fluids given to premature infants may have high aluminum concentrations, including intravenous calcium and phosphorus salts as well as dextrose and mixed parenteral nutrition solutions.[21-23]

Aluminum loading has been observed in patients with normal renal function who receive long-term parenteral nutrition with aluminum-contaminated fluids.[24] The Food and Drug Administration has recommended that concentrations of aluminum in parenteral solutions should not exceed 25 mug/L.[23]

Infant Formulas

Data on the aluminum content of human milk and a variety of infant formulas [25-29] are shown in Table 1. The aluminum levels in all infant formulas are higher than those in human milk. The formulas containing the highest levels of aluminum are those with additives, such as calcium salts and soy protein, which contain aluminum as a contaminant.

There have been conflicting reports on the possibility of aluminum accumulation from infant formulas. One group of investigators have proposed that the aluminum present in infant formulas played a role in the development of aluminum toxicity in two neonates with renal failure.[25] Later, the authors conceded that other unrecognized sources of aluminum, such as intravenous fluids, may have contributed to the excessive aluminum loading in these infants.[30]

Other investigators [20,28] have found no evidence of aluminum accumulation from infant formulas. Formulas for premature infants seem to contain higher levels of aluminum than do standard formulas for term infants.[27,28] Therefore, there is a slightly higher aluminum intake and plasma aluminum concentration in premature infants than in term infants.[27] A provisional tolerable intake recommended by the Food and Agriculture Organization of the United Nations and the World Health Organization [31] is 1 mg/kg per day. Infants fed formulas with even the highest levels of aluminum, 2.35 mg/L, at intakes as high as 200 mL/kg per day would receive an aluminum dose of less than 0.5 mg/kg per day.[26-28] Currently, the data are insufficient to recommend against the use of specialized formulas in premature infants; on the contrary, the nutritional advantages of premature infant formulas clearly outweigh the concern about the higher concentrations of aluminum in these products. However, it seems prudent to seek further reduction in the aluminum levels of infant formulas and to investigate whether aluminum accumulates in the tissue of premature infants fed formulas.

TREATMENT OF ALUMINUM TOXICITY

Deferoxamine administered intravenously has been shown to reduce the body aluminum burden and to ameliorate injury to the bone and brain in adults receiving hemodialysis and peritoneal dialysis.[32] Deferoxamine also has been used successfully to treat aluminum toxicity in children.[33,34]

Although new cases of aluminum intoxication should be preventable in most instances, deferoxamine therapy seems beneficial for those with established aluminum toxicity; however, this therapy is not without hazards. It may cause a number of allergic reactions, including pruritus, wheals, and anaphylaxis. Other adverse effects include dysuria, abdominal discomfort, diarrhea, fever, leg cramps, and tachycardia. Cataracts and neurotoxicity also have been described.[35]

CONCLUSIONS

Dialysis encephalopathy and fracturing osteomalacia, which occur in hemodialysis units that use dialysis fluid contaminated with aluminum, have largely disappeared. This has been accomplished by establishing standards for safe concentrations of aluminum in dialysates. Infants, children, and adults with chronic renal failure who are not receiving dialysis have been shown to be at risk for aluminum intoxication from the oral administration of aluminum-containing phosphate binders. This complication should be avoided with the use of phosphate binders that do not contain aluminum and the use of other measures to control hyperphosphatemia.

A number of substances commonly administered intravenously, including calcium and phosphorus salts and albumin, have high levels of aluminum. Premature infants receiving intravenous fluid therapy may accumulate aluminum and show evidence of aluminum toxicity. Efforts are being made to reduce the levels of aluminum in products added to intravenous solutions; these efforts must continue.

Some infant formulas may contain relatively high concentrations of aluminum. The reported concentrations of aluminum in soy formulas and premature infant formulas are higher than those in other infant formulas. The potential impact of these formulas on the aluminum intake of premature infants and infants with impaired renal function should be recognized, although it is not clear that toxic effects result from the use of the formulas in these situations.

RECOMMENDATIONS

1. Aluminum-containing phosphate binders should not be administered to infants and children with renal failure.

2. Continued efforts should be made to reduce the levels of aluminum in products that are added to intravenous solutions that are used for premature infants and infants and children with renal failure.

3. Continued efforts should be made to reduce the aluminum content of all formulas used for infants, but especially soy formulas and formulas tailored specifically for premature infants.

posted 04-19-2002 09:41 PM            

Aluminum
Aluminum is a relative newcomer to the list of toxic metals, long having been considered harmless. It has been implicated as a contributory cause of Alzheimer's disease. (5) Elevated hair aluminum in children, especially when attended with elevations of lead, have been found to cause decreased visual motor performance. (6)

We routinely recommend hair tests for ADHD children, primarily as a screening test for lead. Rather surprisingly, the most consistent finding is that of elevated hair aluminum.

Aluminum comes from foods cooked or stored in aluminum pans and aluminum foil. Leafy vegetables, rhubarb, and apples cooked in aluminum pans are prone to leach the metal from the pan. Pressure cookers are especially likely to impart metal into the food. Tap water may contain aluminum when it is used in water reservoirs to flocculate silt from the water. Other sources include antacids taken for stomach trouble, some antiperspirants, food additives, and milk substitutes. http://www.oneflesh.org/Child-chap%208.html

Contamination with Aluminum Compounds
Copyright 1996 Leading Edge Research Group, P.O. Box 7530, Yelm, Wasington 98597 USA

General Information
Aluminum is the third most abundant element (8%) in the Earth's crust, exceeded by oxygen (47%) and silicon (28%). Because of its strong affinity to oxygen, aluminum never occurs as a metal in nature but is found only in the form of its compounds, such as alumina. This strong affinity to oxygen also explains why it withstood all attempts to prepare it in its elemental form until well into the 19th century. The metal's name is derived from alumen, the Latin name for alum.

In 1761 the French chemist Guyton de Morveau proposed the name alumine for the base in alum, and in 1787 Antoine Lavoisier identified alumine as the oxide of a then-undiscovered metal.

In 1807 Sir Humphry Davy assigned the name alumium to the metal and later agreed to change it to aluminum. Shortly thereafter, the name aluminium was adopted to conform with the -ium ending of most elements, and this spelling is now in general use throughout the world, except in the United States (where the second i was dropped in 1925) and Italy (where alluminio is used). The role aluminum plays in human physiology is not known. The metal is ingested through food and water. Aluminum has been detected in the brain cells of Alzheimer’s Disease patients. ...

The rest of this article can be found here. http://www.connectcorp.net/~trufax/mercury/alum1.html

And here is the site where I found the article at.

Chemical Manipulation of Consciousness, Behavior,
Health and Evolutionary Potential in the Human Population http://www.connectcorp.net/~trufax/menu/chem.html